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Muscles disorders

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الكلية كلية طب حمورابي     القسم الكلية ذات القسم الواحد     المرحلة 1
أستاذ المادة بشار صاحب خلف الشمري       07/05/2019 09:54:00
Lecture objectives
describe the process of skeletal muscle remodeling and its relevance to atrophy and hypertrophy.
outline the physiology of the neuromuscular junction and describe the pathogenesis and clinical features of myasthenia gravis.
state how neuromuscular transmission is disrupted in botulism and organophosphate poisoning.
describe the pathophysiology of Duchenne muscular dystrophy.
outline the pathophysiology of malignant hyperthermia.
Remodeling of Muscles
Alteration of Neuromuscular Transmission.
Muscular Dystrophies
Other Myopathies
Continual
Replacement of contractile proteins in 2 weeks
Destruction-replacement =atrophy
Replacement-destruction=hypertrophy
Although skeletal muscle has a remarkable capacity to repair and adapt to exercise-induced muscle damage, there is a limit to this capacity for remodelling. The precise impact of repeated bouts of muscle damage and repair, over a number of years, on the remodelling capacity of skeletal muscle is not known. However, there is evidence to suggest that athletes who expose their muscles to rigorous training and racing regimes may exceed the muscles finite capacity for repair and adaptation
Lower motor neuron lesion
Muscle paresis/paralysis, hypotonia/atonia, and hyporeflexia/areflexia are usually seen immediately following an insult. The extensor Babinski reflex is usually absent. Muscle wasting, fasciculations and fibrillations are typically signs of end-stage muscle denervation. Another feature is the segmentation of symptoms - only muscles innervated by the damaged nerves will be symptomatic.


المادة المعروضة اعلاه هي مدخل الى المحاضرة المرفوعة بواسطة استاذ(ة) المادة . وقد تبدو لك غير متكاملة . حيث يضع استاذ المادة في بعض الاحيان فقط الجزء الاول من المحاضرة من اجل الاطلاع على ما ستقوم بتحميله لاحقا . في نظام التعليم الالكتروني نوفر هذه الخدمة لكي نبقيك على اطلاع حول محتوى الملف الذي ستقوم بتحميله .
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